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An Alternative Splicing Switch Regulates Embryonic Stem Cell Pluripotency and Reprogramming [ChIP-Seq]

ChEBI2011-09-14 更新2026-07-21 收录

干细胞多能性调控

可变剪接机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-31006 数据链接链接失效反馈

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An Alternative Splicing Switch Regulates Embryonic Stem Cell Pluripotency and Reprogramming [ChIP-Seq]

应用场景：

创建时间：

2011-09-14

干细胞多能性调控

非编码RNA功能

Table S1. Summary of primer sequences for quantitative real-time PCR in this study. (XLSX 12 kb)

DataCite Commons2024-02-14 更新130

RNA-Seq after knockdown of the core pluripotency transcription factors OCT4, SOX2, and NANOG in TE03 (I3) human embryonic stem cells (hESCs). RNA-Seq after knockdown of the core pluripotency transcription factors OCT4, SOX2, and NANOG in TE03 human embryonic stem cells (hESCs)

干细胞多能性调控

转录后基因表达分析

The OCT4, NANOG and SOX2 transcription factors (TFs) are at the core of the pluripotency program, activating the expression of pluripotency genes and repressing lineage-specific genes, many of which a

NIAID Data Ecosystem30

NANOG amplifies STAT3 activation and they synergistically induce the naïve pluripotent program

干细胞多能性调控

转录因子协同作用

NANOG amplifies STAT3 activation and they synergistically induce the naïve pluripotent program

ChEBI2014-01-06 更新00

AMPK signaling for naïve pluripotency. AMPK signaling for naïve pluripotency

干细胞多能性调控

AMPK信号通路

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem50

Data Sheet 1_ESRP2 constrains EMT plasticity associated with ZEB1 expression in bladder cancer.docx

膀胱癌EMT调控

可变剪接机制

IntroductionEpithelial-to-mesenchymal transition (EMT)-driven phenotypic plasticity promotes bladder cancer (BC) progression and therapy resistance. While EMT has been primarily associated with transc

NIAID Data Ecosystem00

An Alternative Splicing Switch Regulates Embryonic Stem Cell Pluripotency and Reprogramming [ChIP-Seq]

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