GINS4 suppresses ferroptosis by antagonizing p53 acetylation with Snail

Name: GINS4 suppresses ferroptosis by antagonizing p53 acetylation with Snail
Creator: Central South University
Published: 2023-03-21T12:09:37+08:00

integrated proteome resources2026-02-07 收录

铁死亡调控

p53信号通路

数据链接：

https://www.iprox.cn/page/project.html?id=IPX0006128000 数据链接链接失效反馈

官方服务：

资源简介：

GINS4 suppresses ferroptosis by antagonizing p53 acetylation with Snail

提供机构：

Central South University

创建时间：

2023-03-21

A role for p53 in the adaptation to glutamine starvation through the expression of Slc1a3

癌症代谢适应

p53信号通路

Numerous mechanisms to support cells under conditions of transient nutrient starvation have been described. The tumor suppressor protein p53 can contribute to the adaptation of cells to metabolic stre

NIAID Data Ecosystem50

STING aggravates ferroptosis-dependent myocardial ischemia-reperfusion injury by targeting GPX4 for autophagic degradation

心肌缺血再灌注损伤

铁死亡调控

To investigate the potential signal pathways affected by the deletion of the Sting gene in mice during ischemia-reperfusion (IR), we extracted the myocardial tissues of mice 24 hours after ischemia-re

NIAID Data Ecosystem10

Effect of MDM2 overexpression on response to etoposide treatment

癌症药物反应

p53信号通路

We aimed to study the effect of non-mutational p53 inactivation on the response of hematopoietic progenitor cells to genotoxic treatment. In order to study this in vitro, we generated HSPC lines by en

NIAID Data Ecosystem00

Ribo-Seq experiments in HCT116 WT and TP53-/- cells upon neocarzinostatin treatment. Ribo-Seq experiments in HCT116 WT and TP53-/- cells upon neocarzinostatin treatment

翻译调控机制

p53信号通路

The transcription factor p53 exerts its tumor suppressive effects through transcriptional activation of numerous target genes controlling cell cycle arrest, apoptosis, cellular senescence and DNA repa

NIAID Data Ecosystem40

CRISPR activation screens identify the SWI-SNF ATPases as suppressors of ferroptosis [NGS3790_scATAC]

铁死亡调控

单细胞ATAC-seq

Ferroptosis is an iron-dependent cell death mechanism characterized by an accumulation of toxic lipid peroxides and membrane rupture. The glutathione dependent enzyme, GPX4 (glutathione peroxidase 4),

NIAID Data Ecosystem10

GINS4 suppresses ferroptosis by antagonizing p53 acetylation with Snail

资源简介：

相关数据集