首页/数据集/RIP-Seq of MCF-7 cells cultured with senescent neutrophils-derived exosomes, with or without FTO silencing.

RIP-Seq of MCF-7 cells cultured with senescent neutrophils-derived exosomes, with or without FTO silencing.

NIAID Data Ecosystem2026-03-13 收录

RNA修饰调控

乳腺癌基因表达

数据链接：

https://www.ncbi.nlm.nih.gov/sra/SRP371825 数据链接链接失效反馈

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资源简介：

The study aims to explore the mechanism by which exosomal piR-17560 upregulates ZEB1 expression via FTO-mediated m6A modification.

应用场景：

创建时间：

2022-04-25

乳腺癌基因表达

癌症异种移植模型

RNA was isolated from ectopically sFRP1-expressing MDA-MB-231 cells and control MDA-MB-231 cells and as well from tumor lysates arising from these cells as nude mouse xenograft. Gene expression profi

NIAID Data Ecosystem110

EP3

乳腺癌基因表达

RNA测序分析

In the study presented here POSH2 was overexpressed in the MCF10AT breast cancer cells. RNA sequencing profiling of mRNA levels was performed in the control and overexpression cells.

NIAID Data Ecosystem80

Nuclear m6A Reader YTHDC1 suppresses proximal Alternative Polyadenylation sites by Interfering with the 3' Processing Machinery

RNA修饰调控

mRNA前体加工

N6-methyladenosine (m6A) and alternative polyadenylation (APA) are important regulators of gene expression in eukaryotes. Recently, it was found that m6A is closely related to APA. However, the molecu

NIAID Data Ecosystem30

Nuclear 2'-O-methylation regulates RNA splicing through its binding protein FUBP1 (CLIP-seq and PAR-CLIP)

RNA修饰调控

RNA剪接

2'-O-methylation (Nm) is an abundant RNA modification exists on different mammalian RNA species. However, potential Nm recognition by proteins has not been extensively explored. Here, we employed RNA

NIAID Data Ecosystem80

Additional file 10 of The LINC00623/NAT10 signaling axis promotes pancreatic cancer progression by remodeling ac4C modification of mRNA

癌症分子机制

RNA修饰调控

Additional file 10: Table S10. The predicted downstream target genes of NAT10 in BxPC-3 cells.

Figshare2024-02-07 更新50