Supplemental Material for Cao et al., 2021
收藏资源简介:
Figures include Figure 1-5. Figure 1 showed that <i>Brpf1</i> knockdown led to a mild reduced trend on the differentiation of MGE-derived GABAergic interneurons into PV<sup>+</sup> interneurons. Figure 2 showed that <i>Brpf1</i> knockdown had no significant effect on the dendritic morphology of MGE-derived GABAergic interneurons. Figure 3 showed that <i>Brpf1</i> deficiency led to attenuated inhibitory neurotransmission of MGE-derived GABAergic interneurons. Figure 4 showed that <i>Brpf1</i> deficiency had no effect on the neuronal migration of MGE-derived GABAergic interneurons <i>in vivo</i>. Figure 5 showed that <i>Brpf1</i> deficiency led to decreased <i>Map2k7</i> expression in MGE-derived GABAergic interneurons revealed by mRNA-Seq. Supplementary materials include Reagent Table and Table S1. Table S1 contains DESeq2 output and the FPKM values of genes. All reagents and software were listed in the Reagent Table.



