a Group 1, Single copy HPV integration with episomal components; b Group 2, Single copy HPV integration without episomal components; c Group 3, Multicopy tandem repeated integration; d Group 4, low HP

NIAID Data Ecosystem20

Additional file 5: Table S3. of Emerging role of mutations in epigenetic regulators including MLL2 derived from The Cancer Genome Atlas for cervical cancer

宫颈癌基因组学

表观遗传分析

Mutated driver genes and variants in patients. (XLS 99 kb)

Figshare2017-04-09 更新20

Combined arrayCGH and SNP-loss of heterozygosity analysis in cervical cancer. Homo sapiens

宫颈癌基因组学

多组学数据整合

BACKGROUND: Cervical carcinoma develops as a result of multiple genetic alterations. Different studies investigated genomic alterations in cervical cancer mainly by means of metaphase comparative geno

NIAID Data Ecosystem40

Comprehensive analysis of HPV integration in cervical cancer identifies some new genomic hot spots and tumor-relevant genes

HPV整合

宫颈癌基因组学

The exact mechanism for integration, and the role of integration in stimulating cancer progression, is not fully characterized. Given this need, the goal of this study was to search for possible commo

NIAID Data Ecosystem20