A preclinical PoleP286R model of ultramutated endometrial cancer
收藏NIAID Data Ecosystem2026-03-12 收录
官方服务：
资源简介：
characterize ultramuations caused by Pole P296R in endometrial cancer
应用场景：
创建时间：
2021-06-30
相关数据集
MOF forms a positive feedback regulation with ERα to suppress endometrial cancer progression. MOF forms a positive feedback regulation with ERα to suppress endometrial cancer progression
MOF is a histone acetyltransferase specific for H4K16 acetylation. It has been demonstrated that MOF is lower expressed in series of human cancers. However, the molecular mechanism underlying the deta
NIAID Data Ecosystem90
Supplementary Table 2 from SIX1 Regulates Aberrant Endometrial Epithelial Cell Differentiation and Cancer Latency Following Developmental Estrogenic Chemical Exposure
Table S2A: 2,856 DEGs CON-Six1d/d versus CON-Six1+/+ Table S2B: GO Categories from 2,856 DEGs CON-Six1d/d versus CON-Six1+/+ Table S2C: NextBio Bioset overlap with KO Atlas from 2,856 DEGs CON-Six1d/d
NIAID Data Ecosystem70
Breeding trial summary.
Chromatin remodeling plays an integral part in endometrial homeostasis through its roles in the maintenance of cell identity, epithelial integrity, and prevention of endometrial disease. Chromodomain-
NIAID Data Ecosystem30
SALL4 as an Epithelial-Mesenchymal Transition and Drug Resistance Inducer through the Regulation of c-Myc in Endometrial Cancer
SALL4 plays important roles in the development and progression of many cancers. However, the role and molecular mechanism of SALL4 in endometrial cancer remain elusive. In the present research, we hav
NIAID Data Ecosystem80
Chromatin Immunoprecipitation(ChIP)-seq of TCF19 in endometrial cancer cells
In our study, TCF19 was found to be upregulated in microsatellite instability (MSI) endometrial cancer and was associated with poor prognosis and immune exhaustion signature. Elevated TCF19 enhanced
NIAID Data Ecosystem50
