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CircHIPK3 and FMRP compete for direct binding to BRCA1 mRNA controlling its protein levels and DNA damage response

NIAID Data Ecosystem2026-05-02 收录

BRCA1mRNA调控

DNA损伤修复机制

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE246226 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2024-09-13

冷冻电镜结构解析

DNA损伤修复机制

Cryo-EM structure of the RecBCD Chi recognised complex Descriptor: DNA fork substrate, RecBCD enzyme subunit RecB, RecBCD enzyme subunit RecC, ... Authors: Cheng, K, Wilkinson, M, Wigley, D.B. Deposit

Protein Data Bank Japan2024-05-22 更新60

Removal of heat-sensitive clustered damaged DNA sites is independent of double-strand break repair

DNA损伤修复机制

热敏感簇状损伤位点

Abstract DNA double-strand breaks (DSBs) are the most deleterious lesions that can arise in cells after ionizing radiation or radiometric drug treatment. In addition to prompt DSBs, DSBs may also be p

NIAID Data Ecosystem50

Crystal structure of PCNA in complex with DNA polymerase kappa fragment

蛋白质晶体结构解析

DNA损伤修复机制

Crystal structure of PCNA in complex with DNA polymerase kappa fragment Descriptor: DNA polymerase kappa, Proliferating cell nuclear antigen, SULFATE ION, ... Authors: Hishiki, A, Hashimoto, H, Hanafu

Protein Data Bank Japan2024-10-23 更新40

Polymerase ? and mismatch repair cooperatively determine the mutagenicity of a methylating agent

DNA损伤修复机制

甲基化剂致突变性

We characterised genome-wide mutations induced by the O6-dG-methylating agent N-methyl-N'-nitro-N-nitrosoguanidine (MNNG) in mouse embryonic fibroblasts, deficient for the Pol ? catalytic subunit REV3

NIAID Data Ecosystem50

Roles for the Transcription Elongation Factor NusA in Both DNA Repair and Damage Tolerance Pathways in Escherichia coli. Escherichia coli

DNA损伤修复机制

转录偶联修复

Here we report our observations that have led us to propose that the transcription elongation factor NusA promotes a novel class of transcription-coupled repair (TCR) in addition to its previously pro

NIAID Data Ecosystem40

CircHIPK3 and FMRP compete for direct binding to BRCA1 mRNA controlling its protein levels and DNA damage response

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