Mettl3-mediated mRNA m6A methylation promotes dendritic cell activation
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2019-05-01
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Data_Sheet_1_Methyltransferase 3 Mediated miRNA m6A Methylation Promotes Stress Granule Formation in the Early Stage of Acute Ischemic Stroke.PDF
The modification of methyltransferase-like (METTL) enzymes plays important roles in various cellular responses by regulating microRNA expression. However, how m6A modification is involved in stress gr
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MeRIP, RIP and RNA Sequencing of the H1299 stable cell lines which the endogenous YTHDF2 was silenced by a short hairpin RNA targeting YTHDF2 3’UTR (shYTHDF2) and then re-expressed Flag-tagged YTHDF2-WT orYTHDF2-K571R
SUMOylation affects many aspects of target proteins such as activity, stability, localization and protein-protein interactions. We have found that SUMOylation of YTHDF2 increased its binding activity
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Depletion of m6A methylation by conditional Mettl3 knockout in Ascl1-induced neurons
Depletion of m6A methylation by conditional Mettl3 knockout in Ascl1-induced neurons
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Transcriptome sequencing of IPEC-J2 cells between shWTAP and shCtrl group
N6-methyladenosine (m6A) modification is the most prevalent chemical mark in mammalian mRNAs. Generally, m6A modification is deposited to RNAs by the m6A methyltransferase complex, including WTAP. Nev
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File S1 - Molecular Characterization of Antigen-Peptide Pulsed Dendritic Cells: Immature Dendritic Cells Develop a Distinct Molecular Profile when Pulsed with Antigen Peptide
Contents include: Table S1. Significant genes in mDC induced by CD40L/TNFα compared with piDC. Table S2. Significant genes in mDC induced by cytokine cocktail compared with piDC. Table S3. Bioinformat
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