Bulk RNA-seq of NOX2 difieicent Kupffer cells in ASH-induced mice

NIAID Data Ecosystem2026-05-02 收录

酒精性肝病机制

多组织巨噬细胞转录组

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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1156317 数据链接链接失效反馈

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This study aims to investigate the characteristic changes in NOX2-deficient Kupffer cells after chronic and binge ethanol feeding.

创建时间：

2024-09-04

Chronic Alcohol Consumption Alters micro-RNA Radiation Responses in the Mouse Liver

酒精性肝病机制

应激miRNA调控

Accumulating evidence suggests that lifestyle-related factors may influence radiation responses and the resulting cancer risks through epigenetic mechanisms, such as miRNA regulations. Chronic alcohol

NIAID Data Ecosystem60

The Integrated "Multiomics" Landscape at Peak Injury and Resolution From Alcohol-Associated Liver Disease

酒精性肝病机制

肝脏转录组

To investigate the the liver transcriptome at peak injury and during early and late resolution from alcohol-induced liver injury in mice Comparative gene expression profiling analysis of RNA-seq data

NIAID Data Ecosystem40

Effect of depletion of hepatocellular STAT3 on alcohol-treated liver tissue. Effect of depletion of hepatocellular STAT3 on alcohol-treated liver tissue

酒精性肝病机制

STAT3信号通路

To investigate the influence of hepatocellular STAT3 activation on liver under alcohol condiction, mice of Stat3 condictional knock-out in hepatocytes were treated by alcohol for 2 weeks. Overall desi

NIAID Data Ecosystem20

Notch Pathway Activation Contributes to Inhibition of C2C12 Myoblast Differentiation by Ethanol

酒精性肝病机制

Notch信号通路

The loss of muscle mass in alcoholic myopathy may reflect alcohol inhibition of myogenic cell differentiation into myotubes. Here, using a high content imaging system we show that ethanol inhibits C2C

Figshare2016-01-18 更新40

DataSheet1_Alcohol Abstinence Rescues Hepatic Steatosis and Liver Injury via Improving Metabolic Reprogramming in Chronic Alcohol-Fed Mice.docx

酒精性肝病机制

戒酒治疗效应

Background: Alcoholic liver disease (ALD) caused by chronic ethanol overconsumption is a common type of liver disease with a severe mortality burden throughout the world. The pathogenesis of ALD is co

NIAID Data Ecosystem30

Bulk RNA-seq of NOX2 difieicent Kupffer cells in ASH-induced mice

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