E-GEOD-54785

NIAID Data Ecosystem2026-03-08 收录

去乙酰化酶功能

结肠炎机制

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https://www.omicsdi.org/dataset/atlas-experiments/E-GEOD-54785 数据链接链接失效反馈

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资源简介：

The acetylome regulators Hdac1 and Hdac2 differently modulate intestinal epithelial cell dependent homeostatic responses in experimental colitis

创建时间：

2015-08-04

Table_1_A Class 1 Histone Deacetylase as Major Regulator of Secondary Metabolite Production in Aspergillus nidulans.pdf

真菌代谢调控

去乙酰化酶功能

An outstanding feature of filamentous fungi is their ability to produce a wide variety of small bioactive molecules that contribute to their survival, fitness, and pathogenicity. The vast collection o

NIAID Data Ecosystem70

Most specific biological processes affected by KDAC inactivation.

表观遗传调控

去乙酰化酶功能

Most specific biological processes affected by KDAC inactivation.

NIAID Data Ecosystem20

The primer sets for qPCR.

结肠炎机制

qPCR基因表达验证

Background Ulcerative colitis (UC) is a recurrent inflammatory condition of the bowel with a multifaceted pathogenesis, including programmed cell death, oxidative stress, and immune-mediated inflammat

NIAID Data Ecosystem10

Intestinal epithelial deletion of the glucocorticoid receptor NR3C1 alters expression of inflammatory mediators and barrier function

结肠炎机制

糖皮质激素受体功能

Glucocorticoids (GCs) are important hormones involved in the regulation of multiple physiologic functions. GCs are also widely used in anti-inflammatory/immunosuppressant drugs. GCs are synthesized by

NIAID Data Ecosystem50

Stem-like T cells are associated with the pathogenesis of ulcerative colitis in humans [scRNAseq_Human]

结肠炎机制

干细胞研究

To understand the role of T cells in the pathogenesis of ulcerative colitis (UC), we analyzed colonic T cells isolated from UC patients and controls. We identified colonic CD4+ and CD8+ 32 T lymphocyt

NIAID Data Ecosystem10

E-GEOD-54785

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