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IGF2BP2-regulated alternative splicing of NFIC causes excessive granulosa cell proliferation in PCOS. IGF2BP2-regulated alternative splicing of NFIC causes excessive granulosa cell proliferation in PCOS

NIAID Data Ecosystem2026-03-13 收录

PCOS分子病理学

细胞增殖调控

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA789474 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

应用场景：

创建时间：

2021-12-16

细胞增殖调控

肺病分子机制

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

NIAID Data Ecosystem40

Code and data from "Mother cells control daughter cell proliferation in intestinal organoids to minimize proliferation fluctuations"

类器官研究

细胞增殖调控

Includes the microscopy images, cell tracking data and scripts used in the publication Huelsz-Prince, Guizela, et al. "Mother cells control daughter cell proliferation in intestinal organoids to minim

NIAID Data Ecosystem30

Expression data from human corneal endothelial cells. Homo sapiens

内皮细胞基因表达

细胞增殖调控

Human corneal endothelial cells (HCEC) form a monolayer by adhering tightly through their intercellular adhesion molecules. Located at the posterior corneal surface, they maintain corneal translucency

NIAID Data Ecosystem20

Different CAP parameter (output voltage and resistance value) effects for MSC growth in vitro.

细胞电刺激

细胞增殖调控

Note: “+” means positive effects (i.e., non-inhibited or increased cell proliferation when compared to untreated group) for MSCs; “−” means negative inhibited effects (i.e., decreased cell proliferati

Figshare2015-12-02 更新30

Table 3_Circ-104792/miR-133a/Bcl-xL influences the proliferation and function of human trophoblastic and decidual stromal cells involved in recurrent abortion disease.xls

复发性流产机制

细胞增殖调控

BackgroundThe circRNA-miRNA axis is critically implicated in the pathogenesis of recurrent spontaneous abortion (RSA) by modulating trophoblast and decidual stromal cell functions. This study investig

NIAID Data Ecosystem20

IGF2BP2-regulated alternative splicing of NFIC causes excessive granulosa cell proliferation in PCOS. IGF2BP2-regulated alternative splicing of NFIC causes excessive granulosa cell proliferation in PCOS

资源简介：

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