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The role of endogenous Smad7 in regulating macrophage phenotype following myocardial infarction. The role of endogenous Smad7 in regulating macrophage phenotype following myocardial infarction

NIAID Data Ecosystem2026-03-13 收录

心肌梗死

巨噬细胞调控

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA783034 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

应用场景：

创建时间：

2021-11-23

心肌梗死

缺氧

Background/Aims: Patients with myocardial infarction and hypoxemia require supplemental oxygen. However, the current therapeutic paradigm is contradicted by several recent studies in whi

DataCite Commons2025-05-01 更新90

Sex Differences in the Effects of COPD on Incidence and Outcomes of Patients Hospitalized with ST and Non-ST Elevation Myocardial Infarction: A Population-Based Matched-Pair Analysis in Spain (2016-2018)

慢性阻塞性肺疾病

心肌梗死

Global Health Data Exchange ()40

Relationship Between Different Risk Factor Patterns and Follow-Up Outcomes in Patients With ST-Segment Elevation Myocardial Infarction

心血管疾病

心肌梗死

Relationship Between Different Risk Factor Patterns and Follow-Up Outcomes in Patients With ST-Segment Elevation Myocardial Infarction

Global Health Data Exchange ()20

Supplementary Material for: The Spleen Promotes the Secretion of CCL2 and Supports an M1 Dominant Phenotype in Hepatic Macrophages During Liver Fibrosis

肝纤维化

巨噬细胞调控

Background/Aims: Liver fibrosis is a complex process of tissue remodeling in response to injury. Hepatic macrophages have been identified as a key player in this process. As the largest

DataCite Commons2020-08-28 更新60

table_3_Glucocorticoids Impair Phagocytosis and Inflammatory Response Against Crohn’s Disease-Associated Adherent-Invasive Escherichia coli.PDF

鱼类肠道免疫病理学

巨噬细胞调控

Crohn’s disease (CD) is a chronic inflammatory bowel disorder characterized by deregulated inflammation triggered by environmental factors. Notably, adherent-invasive Escherichia coli (AIEC), a bacter

NIAID Data Ecosystem30

The role of endogenous Smad7 in regulating macrophage phenotype following myocardial infarction. The role of endogenous Smad7 in regulating macrophage phenotype following myocardial infarction

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