Hypoxia is a hallmark of the tumor microenvironment that profoundly alters the cellular metabolism and epigenetic regulation. In this study, we investigated how oxygen limitation reprograms histone me

Figshare2025-11-26 更新50

Dynamic interplay between sortilin and syndecan-1 has a role in the metabolic switch during prostate cancer progression. Dynamic interplay between sortilin and syndecan-1 has a role in the metabolic switch during prostate cancer progression

癌细胞重编程

前列腺癌分子病理学

Prostate cancer development and progression relies on the programming of glucose and lipid metabolism, and this involves alterations in androgen receptor expression and signalling. Androgen deprivatio

NIAID Data Ecosystem20

PDSS2 and Prostate

前列腺癌分子病理学

基因表达预后预测

In this study we tested the notion that PDSS2 expression is greater in non-neoplastic prostate tissue than in neoplastic tissue and that PDSS2-positive neoplasms are relatively indolent due to the mai

Mendeley Data2021-02-11 更新60

Amplification and overexpression of vinculin are associated with increased tumor cell proliferation and progression in advanced prostate cancer. Homo sapiens

前列腺癌分子病理学

肿瘤进展机制

We analyzed the 10q22 amplification in advanced prostate cancer and subjected the genes located in the common amplified region to an RNAi screen. We found vinculin as the most promising candidate gene

NIAID Data Ecosystem40

Table S9 from LHX2 Rewires the Metabolic and Epigenetic Landscape to Drive Tumor Progression in Prostate Cancer

资源简介：

相关数据集