m6A methylation regulates the fate of endogenous retrovirus transcripts

NIAID Data Ecosystem2026-03-13 收录

RNA表观遗传学

内源性逆转录病毒

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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE145616 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

创建时间：

2021-10-26

Table_9_Roles of m5C RNA Modification Patterns in Biochemical Recurrence and Tumor Microenvironment Characterization of Prostate Adenocarcinoma.xlsx

前列腺癌分子预后

RNA表观遗传学

BackgroundProstate cancer is the second most common cancer with a high risk of biochemical recurrence (BCR) among men. Recently, 5-methylcytosine (m5C) modification has attracted more attention as a n

NIAID Data Ecosystem30

The N6-methyladenosine RNA landscape in aged mouse hippocampus [meRIP-seq]

RNA表观遗传学

神经衰老机制

Aged brain is associated with an inevitable decline in cognitive functions and increased vulnerability to neurodegenerative disorders. Multiple molecular hallmarks have been associated with the aging

NIAID Data Ecosystem20

N6-methyladenosine (m6A) recruits and repels proteins to regulate mRNA homeostasis. Homo sapiens

RNA表观遗传学

RNA稳定性调控

RNA modifications are integral to regulation of RNA metabolism. One such abundant mRNA modification is m6A, which impacts various aspects of RNA metabolism including splicing, transport and degradatio

NIAID Data Ecosystem10

Analysis of m6A methylome in CHAPIR overexpressed heart [MeRIP]

RNA表观遗传学

心脏疾病分子机制

To elucidate the molecular mechanism by which cardiac-hypertrophy-associated piRNA (CHAPIR) regulates m6A modification, we performed m6A methylated RNA immunoprecipitation sequencing (MeRIP-seq) in co

NIAID Data Ecosystem20

Table_1_Transcriptome and N6-Methyladenosine RNA Methylome Analyses in Aortic Dissection and Normal Human Aorta.docx

主动脉夹层分子机制

RNA表观遗传学

Objective: To investigate the N6-methyladenosine (m6A) modification and the expressions of the m6A regulatory genes in the acute aortic dissection (AD). Methods: MeRIP-seq and RNA-seq experiments of a

NIAID Data Ecosystem10

m6A methylation regulates the fate of endogenous retrovirus transcripts

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