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Inhibition of proliferation and promotion of apoptosis and DNA damage by the targeting of STAT3 by miR-125a-5p in irradiated mouse intestinal epithelial cells.
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创建时间：
2023-02-02
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Figure S5
Samples of primary fibroblasts M. m. musculus (PWK) and M. spretus (STF) were transferred into X-RAD 320 X-Ray Biological Irradiator and treated with 10 Gy, 15 Gy, 20 Gy, and 25 Gy. Same XGal scoring
NIAID Data Ecosystem60
Table1_Chang qing formula ameliorates colitis-associated colorectal cancer via suppressing IL-17/NF-κB/STAT3 pathway in mice as revealed by network pharmacology study.XLSX
Colitis-associated colorectal cancer (CAC) is a specific type of colorectal cancer (CRC) with high mortality and morbidity, the chronic inflammation in the intestinal mucosal is the characteristic of
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CDYL2 epigenetically regulates MIR124 to control NF-kB/STAT3-dependent breast cancer cell plasticity [RNA-seq]
Epigenetic deregulation of gene transcription is central to cancer cell plasticity and malignant progression, but remains poorly understood. We found that the uncharacterized epigenetic factor Chromod
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Resistance to Spindle inhibitors in Glioblastoma Depends on STAT3 and Therapy Induced Senescence. Resistance to Spindle inhibitors in Glioblastoma Depends on STAT3 and Therapy Induced Senescence
While mitotic spindle inhibitors specifically kill proliferating tumor cells without the toxicities of microtubule poisons, resistance has limited their clinical utility. Treating glioblastomas with t
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Genome-wide ChIP-Seq analysis of genomic Stat3 binding sites in murine gastric tumors following stimulation with IL6 or IL11 (MACS). Mus musculus
The latent transcription factor STAT3 is a point of convergence for numerous oncogenic signaling pathways and is excessively activated in the majority of human epithelial cancers. STAT3 promotes many
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