Introduction: Neointimal hyperplasia after percutaneous coronary intervention remains a major determinant of in-stent restenosis (ISR). The extent of mechanical vessel injury correlates with ISR. A ne
A MS-Excel notebook of several spreadsheets pertaining to Figs 1–9 and Table 1, including ANOVA analysis, means, standard deviations, and charts/graphs, and also an adaptation of Andersen 1958 Table 1
Here we performed single cell transcriptomic analysis of mouse carotid arteries following a ligation injury to investigate SMCs heterogeneity at the single-cell level after vascular injury.
