This dataset contains transcriptomic data of splenic B cells from mice with B cell-specific deletion of Rabggta and control mice. Both Rabggta -cKO mice and WT mice have 4 individuals eac
CAB2 mutant derived from the clinical isolate RimD2210633. CAB2 lacks TDH hemolysins and its T3SS1 is not functional by means of deletion of exsA, a gene encoding the T3SS1 transcription regulator.
Acute Myeloid Leukemia (AML) has been associated with somatic mutations in numerous genes; however, the penetrance of these mutations is low. Here we investigate the contribution of alternative splici