We performed H3K9me2-based ChIP-seq to identify regions of the Drosophila genome that are H3K9me2-depleted due to transgenic neuronal expression of human mutant tau.
Defects of autophagy-lysosomal protein degradation are thought to contribute to the pathogenesis of several neurodegenerative diseases, and the accumulation of aggregation prone proteins such as MAPT/
This file contains raw data for the two MS analyses in the tabs labeled “Protein groups”; analyzed data to calculate relative percent abundances of identified proteins in the tabs labeled “Relative ab