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Antigen Presentation-Independent Reciprocal Immune Modulation by HLA-DRB1 Allelic Epitopes that Associate with Autoimmune Disease Risk or Protection

NIAID Data Ecosystem2026-03-12 收录

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数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE159821 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2021-02-02

COVID-19

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Additional file 2: Table S1. Database accession numbers of individual datasets used and accompanying citations used throughout this study.

DataCite Commons2022-04-15 更新70

Table_9_Identification of the Association Between Toll-Like Receptors and T-Cell Activation in Takayasu’s Arteritis.xlsx

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基因表达生物标志物

To explore the relationships between Toll-like receptors (TLRs) and the activation and differentiation of T-cells in Takayasu’s arteritis (TAK), using real-time fluorescence quantitative polymerase ch

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Table 1_Deciphering the role of IL17RA in psoriasis and chronic mucocutaneous candidiasis: shared pathways and distinct manifestations.xlsx

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炎症通路

IntroductionPsoriasis and chronic mucocutaneous candidiasis (CMC), although distinct in their clinical manifestations, often coexist within specific patient cohorts. Despite this intriguing clinical o

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Development of Ferret Reference Resources and Profiling Assays.

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雪貂

Development of reference resources and assays that are essential for ferret immunogenetics and immune repertoire profiling analysis.

DataCite Commons2025-03-21 更新60

Evolutionarily selected overexpression of the cytokine BAFF enhances mucosal immune response against P. falciparum. Evolutionarily selected overexpression of the cytokine BAFF enhances mucosal immune response against P. falciparum

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A variant of the TNFSF13B gene (BAFF-var) increases the production of the cytokine BAFF, up-regulating humoral immunity and increasing the risk for certain autoimmune diseases. BAFF-var was evolutiona

NIAID Data Ecosystem30

Antigen Presentation-Independent Reciprocal Immune Modulation by HLA-DRB1 Allelic Epitopes that Associate with Autoimmune Disease Risk or Protection

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