Neutrophils drive skin autoinflammation by releasing IL-26

NIAID Data Ecosystem2026-05-01 收录

脓疱型银屑病免疫机制

皮肤炎症转录组学

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE255248 数据链接链接失效反馈

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we profiled the inflammatory transcriptome of skin lesions from pustular psoriasis patients Inflammation-oriented transcriptomics was performed on RNA extracted from skin biopsies using NanoString technologies

创建时间：

2024-03-13

Quantitative Analysis of CXCL4 and TLR8 signaling crosstalk in human primary monocytes by RNA-Seq. Quantitative Analysis of CXCL4 and TLR8 signaling crosstalk in human primary monocytes by RNA-Seq

免疫通路分析

皮肤炎症转录组学

To gain a comprehensive understanding of gene regulation in CXCL4 and TLR8 signaling crosstalk, we treated primary human blood monocytes with CXCL4 and TLR8 ssRNA ligand ORN8L for 6 h and performed tr

NIAID Data Ecosystem40

Additional file 3: Table S3. of RNA sequencing from human neutrophils reveals distinct transcriptional differences associated with chronic inflammatory states

皮肤炎症转录组学

中性粒细胞lncRNA

Long non-coding RNAs identified as expressed in neutrophils. (XLSX 341Â kb)

NIAID Data Ecosystem40

Transcriptome analysis of atopic dermatitis and psoriasis lesional skin. Transcriptome analysis of atopic dermatitis and psoriasis lesional skin

皮肤炎症转录组学

银屑病关节炎转录组学

Purpose: To deeply understand the clinical as well as pathogenetic differences, we have analyzed the genes involved in the epidermal lipid synthesis, epidermal development process and immune responses

NIAID Data Ecosystem30

H3K18Ac and H3K18Cr ChIP-seq and RNA-seq analysis of LPS-stimulated RAW 264.7 cells. Mus musculus

组蛋白修饰

皮肤炎症转录组学

(1) We sought to characterize the genomic profiles of H3K18Ac and H3K18Cr before and after the activation of the LPS-induced inflamatory response to elucidate the role of differential acylation in the

NIAID Data Ecosystem40

Genome-wide transcriptome analysis of human iPSC-derived healty control and schizophrenia cortical interneurons under inflammation conditions.. Genome-wide transcriptome analysis of human iPSC-derived healty control and schizophrenia cortical interneurons under inflammation conditions.

精神分裂症细胞模型

皮肤炎症转录组学

We report changes in gene expression in cortical interneurons under inflammation conditions by genome-wide transcriptome analysis, compared to the previous dataset generated without inflammation condi

NIAID Data Ecosystem30

Neutrophils drive skin autoinflammation by releasing IL-26

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