Treatment-induced tumor dormancy through YAP-mediated transcriptional reprogramming of the apoptotic pathway
收藏NIAID Data Ecosystem2026-03-11 收录
官方服务：
资源简介：
This SuperSeries is composed of the SubSeries listed below. Refer to individual Series
应用场景：
创建时间：
2020-01-21
相关数据集
Additional file 1: of NR1B2 suppress kidney renal clear cell carcinoma (KIRC) progression by regulation of LATS 1/2-YAP signaling
Table S1. Comparison of baseline clinicopathological characteristics based on TCGA. Table S2. Univariate and multivariate Cox proportional hazards analysis of OS from TCGA cohort. Table S3. Comparison
DataCite Commons2024-02-26 更新60
DataSheet5_Three-Dimensionally Printed Ti2448 With Low Stiffness Enhanced Angiogenesis and Osteogenesis by Regulating Macrophage Polarization via Piezo1/YAP Signaling Axis.zip
Previous studies have found that the novel low-elastic-modulus Ti2448 alloy can significantly reduce stress shielding and contribute to better bone repair than the conventional Ti6Al4V alloy. In this
NIAID Data Ecosystem20
Decoding YAP dependent transcription in the liver [HepG2_RNASeq]
In this study we aimed to address how YAP elicits profound biological changes in murine liver, such as proliferation, regeneration and dedifferentiation. We demonstrated that YAP is a master regulator
NIAID Data Ecosystem20
Additional file 3 of Deciphering genetic and nongenetic factors underlying tumour dormancy: insights from multiomics analysis of two syngeneic MRD models of melanoma and leukemia
Additional file 3: Figure S2. Multiomics data analysis of CNV, histones marks, and transcriptomic gene expression data for the indicated exclusively expressed proteins in dormant or parental cells fro
Figshare2024-09-05 更新50
Co-culture of human prostate cancer cells and mouse osteoblasts as a model for tumor dormancy in bone metastasis. Co-culture of human prostate cancer cells and mouse osteoblasts as a model for tumor dormancy in bone metastasis
Prostate cancer (PCa) cells, when disseminated to the bone, may stay quiescent or dormant for years before proliferation into overt metastases. Previous studies suggest that osteoblasts, the bone form
NIAID Data Ecosystem20
