HPV infection results in changes in host gene methylation which, in turn, are thought to contribute to the neoplastic progression of HPV-associated cancers. The objective of this study was to identify
The majority of human cancers become highly dependent on a deregulated MYC oncogene, thus identifying a common mechanism underlying MYC regulation could provide valuable targets for therapy. We show h
Table S4. CDGs associated with significant genome-wide methylation changes in one cancer type. Hyper-methylation is defined as CDGs with pi,k <0.05 and the number of hyper-methylated sites is large
