We identified NFIB as a CARM1 substrate and showed that this transcription factor utilizes CARM1 as a coactivator. Importantly, NFIB harbors both oncogenic and metastatic activities, and is often over
Additional file 3. The list of relevant enriched pathways(p value $$\le 0.005$$ ≤ 0.005 ) obtained by CePa using each cancergene list and the related intersection between the two lists ofenriched path
Supplementary Table 2 shows adaptively sampled mean (ASM) and adaptively sampled 95th percentile size (AS95) and significance of genes in the Lenti-sgTSG19 pools on EML4-ALK-V1 and V3-driven lung tumo
