Sepsis decreases lung SVEP1 expression in a murine model

NIAID Data Ecosystem2026-03-14 收录

脓毒症基因表达

器官疾病分子机制

数据链接：

https://www.omicsdi.org/dataset/biostudies-other/S-BSST942 数据链接链接失效反馈

官方服务：

资源简介：

Basic datas in murine model experiments for SVEP1 and sepsis

创建时间：

2023-01-04

Gene and metabolite time-course response to cigarette smoking in mouse lung and plasma

器官疾病分子机制

代谢组学时间序列分析

Prolonged cigarette smoking (CS) causes chronic obstructive pulmonary disease (COPD), a prevalent serious condition that may persist or progress after smoking cessation. To provide insight into how CS

NIAID Data Ecosystem70

Full blots for DEVELOPMENTAL-CELL-D-24-00333R2

蛋白质表达分析

器官疾病分子机制

Figure 7B Western blot analysis of Nr2f2 over-expression in lung c-Kit+ cells after AAV infection. Figure S4B Western blot analysis showing the effects for inducing DTR expression in lung c-Kit+ cells

NIAID Data Ecosystem60

KCa3.1 K+ Channel Expression and Function in Human Bronchial Epithelial Cells

器官疾病分子机制

上皮细胞离子通道

The KCa3.1 K+ channel has been proposed as a novel target for pulmonary diseases such as asthma and pulmonary fibrosis. It is expressed in epithelia but its expression and function in primary human br

Figshare2016-01-15 更新50

Whole-genome gene expression profiles of non-tumorous human lung tissues: GRNG set

组织基因表达

器官疾病分子机制

We profiled genome-wide gene expression in non-tumorous human lung tissues. The overall goal of this project is to improve our molecular understanding of various lung diseases including lung cancer an

NIAID Data Ecosystem70

HDMA Fragment Files + Count Matrices Lung Part2

单细胞多组学

器官疾病分子机制

This is the ATAC fragment files and RNA count matrices for HDMA (Liu*, Jessa*, Kim*, Ng* et al, bioRxiv 2025)

NIAID Data Ecosystem60