To investigate the potential pathogenic role of SLC25A3 defects in myocardial hypertrophy, we performed RNA sequencing on WT and KO cell groups after culturing hiPSC-CMs.
Filtered variant call format (VCF) data of Jinbuol (JBO, female), Samgwang (SG, male), two RIL individuals (JSRIL1 and JSRIL2), Nipponbare1, IndicaHR12, Kitaake, and Kasalath using resequencing data p
