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资源简介:
Metastatic Breast Cancer patient tumor mass whole exome and transcriptome sequencing
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创建时间:
2017-09-17
相关数据集
Mutant-SETBP1 activates transcription of Myc programs to accelerate CSF3R-driven myeloproliferative neoplasms. Mutant-SETBP1 activates transcription of Myc programs to accelerate CSF3R-driven myeloproliferative neoplasms
Colony stimulating factor 3 receptor (CSF3R) mutations lead to JAK pathway activation and are the molecular hallmark of chronic neutrophilic leukemia (CNL). Approximately half of CNL patients also hav
NIAID Data Ecosystem110
Subclonal evolution of four ER+ breast cancers determined by WGS and scRNA-Seq
We analyzed the subclonal evolution of four ER+ breast cancers over 2-15 years using whole-genome sequencing (WGS) and single-cell RNA-Seq (scRNA-Seq). For each patient we analyzed 2-6 cancer samples,
NIAID Data Ecosystem80
Table S13 from Multiomics Reveals the Immunologic Features and the Immune Checkpoint Blockade Potential of Colorectal Medullary Carcinoma
Supplementary Table 13. GSEA results between MeC and nMeC using hallmark gene sets from the MSigDB database
NIAID Data Ecosystem40
POG_patient_27216 - samples
Genome and transcriptome sequence data from a metastatic breast cancer patient, generated as part of the BC Cancer Agency's Personalized OncoGenomics (POG) studyEGA dataset EGAD00001003033
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Multiomics of three distinct hematological malignancies in a patient reveals their common origin from clonal hematopoietic stem cells. Multiomics of three distinct hematological malignancies in a patient reveals their common origin from clonal hematopoietic stem cells
Concomitant multiple hematological malignancies are rare and challenging to diagnose. They represent a unique model to explore the multistep process of oncogenesis. Here, we report the unique case of
NIAID Data Ecosystem40



