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PDGFRα-induced stromal maturation is required to restrain postnatal intestinal epithelial stemness and promote defense mechanisms.

NIAID Data Ecosystem2026-03-13 收录

肠道干细胞调控

上皮-间质相互作用

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE200365 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2022-08-04

肠道干细胞调控

脂质代谢遗传学

Objective: The intestine occupies the critical interface between cholesterol absorption and excretion. Despite this, surprisingly little is known about the role of de novo cholesterol synthesis in thi

NIAID Data Ecosystem40

Fig 2 Data.

肠道干细胞调控

基底膜组织修复

The basement membrane (BM) plays critical roles in stem cell maintenance and activity control. Here we show that chondroitin sulfate (CS), a major component of the Drosophila midgut BM, is required fo

Figshare2025-05-09 更新20

Effect of depletion of SOD1 on gene expression of organoid culture

肠道干细胞调控

基因表达谱分析

To investigate the function of SOD1 in regulation of ISC growth, we established intestinal organoids from tamoxifen–inducible intestinal epithelial cell (IEC)–specific Sod1 knockout (Sod1f/f; Vil-creE

NIAID Data Ecosystem20

Diet-induced alteration of intestinal stem cell function underlies obesity and pre-diabetes in mice

肠道干细胞调控

单细胞转录组学

This dataset consists of single-cell RNA-seq (10X) data from small intestinal crypts of healthy and HFHSD induced obese mice Foxa2-FVF/FVF mice and from small intestinal villi of healthy and HFHSD ind

NIAID Data Ecosystem30

A modified system to promote stemness of mouse intestinal stem cells by activating Nrf2 and α2-adrenergic receptor signaling pathway

肠道干细胞调控

辐射损伤修复

Intestinal stem cells (ISCs) maintain epithelial homeostasis through continuous self-renewal and differentiation, but their regulatory mechanisms remain incompletely understood. Using a simplified cul

中国科学数据2026-04-03 更新20

PDGFRα-induced stromal maturation is required to restrain postnatal intestinal epithelial stemness and promote defense mechanisms.

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