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2021-03-03
相关数据集
Additional file 1 of Establishment and characterization of canine mammary tumoroids for translational research
Additional file 1: Fig. S1. Histology and receptor status (ER, PR, HER2) of the 6 canine mammary tumors used in the study.
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DataSheet2_Wenfei Buqi Tongluo Formula Against Bleomycin-Induced Pulmonary Fibrosis by Inhibiting TGF-β/Smad3 Pathway.xlsx
Pulmonary fibrosis (PF) is the end stage of various chronic and progressive interstitial lung diseases. TGF-β, a profibrotic cytokine, can promote epithelial–mesenchymal transition (EMT), extracellula
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HGF Expressing Stem Cells in Usual Interstitial Pneumonia Originate from the Bone Marrow and Are Antifibrotic
BackgroundPulmonary fibrosis may result from abnormal alveolar wound repair after injury. Hepatocyte growth factor (HGF) improves alveolar epithelial wound repair in the lung. Stem cells were shown to
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ChIP-seq to reveal the genomic binding landscape of Smad3 in renal tubular cells TCMK1
Smad3 is an important downstream transcriptional factor of TGF-Ã signaling in the pathogenesis of renal fibrosis. Understanding the target genes is essential to decode the mechanism underlying Smad3-
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RNA-sequence of epithelial-specific active mTOR transgenic mouse lung. RNA-sequence of epithelial-specific active mTOR transgenic mouse lung
The purpose of this study is to identify the roles of epithelial active mTOR in lung fibrosis. We generated Sftpc-mTOR transgeneic mice, in which active mTOR is conditionally expressed in alveolar epi
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