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Figure 7A
A. MRC5 cells were plated on coverslips and infected with TB40/E-mCh), UL88-STOP-mCh, or UL88-Rev-mCh at MOI of 0.05. Cells were fixed day 7 and day 9 and subjected to IF analysis: DAPI (blue)
DataCite Commons2025-06-13 更新80
Apoptotic exosome-like vesicles regulate endothelial gene expression, inflammatory signaling, and function through the NF-κB signaling pathway.. Apoptotic exosome-like vesicles regulate endothelial gene expression, inflammatory signaling, and function through the NF-κB signaling pathway.
Persistent endothelial injury promotes maladaptive responses by favoring the release of factors leading to perturbation in vascular homeostasis and tissue architecture. Caspase-3 dependent death of mi
NIAID Data Ecosystem60
Effects of Inhibition of CDK8/19 Mediator Kinase by Senexin B in HEK293 cells treated with or without TNF-alpha
We have investigated the effects of CDK8/19 inhibitor Senexin B on NF?B-mediated transcription, induced by a canonical NF?B activator, TNFa. Overall design: HEK293 cells were pre-treated with or witho
NIAID Data Ecosystem50
Table_1_Vildagliptin Reduces Stenosis of Injured Carotid Artery in Diabetic Mouse Through Inhibiting Vascular Smooth Muscle Cell Proliferation via ER Stress/NF-κB Pathway.xls
Dipeptidyl peptidase-4 (DPP-4) inhibitors are novel anti-hyperglycemic drugs for type 2 diabetes. It has been reported that DDP-4 inhibitor could exert pleiotropic effects on cardiovascular system. Th
NIAID Data Ecosystem70
Evolutionary Conserved Regulation of HIF-1β by NF-κB
Hypoxia Inducible Factor-1 (HIF-1) is essential for mammalian development and is the principal transcription factor activated by low oxygen tensions. HIF-α subunit quantities and their associated acti
NIAID Data Ecosystem60
