首页/数据集/Suppression of poised oncogenes by ZMYND8 promotes chemo-sensitization. Suppression of poised oncogenes by ZMYND8 promotes chemo-sensitization

Suppression of poised oncogenes by ZMYND8 promotes chemo-sensitization. Suppression of poised oncogenes by ZMYND8 promotes chemo-sensitization

NIAID Data Ecosystem2026-03-11 收录

乳腺癌耐药性

靶向表观遗传疗法

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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA606181 数据链接链接失效反馈

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In this study, we have shown how ZMYND8, a putative chromatin reader, chemosensitizes, chemo-resistant breast cancer cells, by modulating poised oncogenes. Overall design: Epigenetic therapy to treat chemo-resistant breast cancer

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创建时间：

2020-02-12

乳腺癌耐药性

WEE1抑制剂抗癌

Despite the success of antiestrogens in extending overall survival of patients with estrogen receptor positive (ER+) breast tumors, resistance to these therapies is prevalent. ER+ tumors that progress

NIAID Data Ecosystem60

DataSheet_1_A Phase II Trial of the Double Epigenetic Priming Regimen Including Chidamide and Decitabine for Relapsed/Refractory Acute Myeloid Leukemia.docx

急性髓系白血病治疗

靶向表观遗传疗法

ObjectiveTo explore the role of chidamide, decitabine plus priming regimen in the salvage treatment of relapsed/refractory acute myeloid leukemia. MethodsA clinical trial was conducted in relapsed/ref

NIAID Data Ecosystem20

Effect of cold atmospheric plasma (CAP) in MCF-7 breast cancer cells [dataset 1]. Effect of cold atmospheric plasma (CAP) in MCF-7 breast cancer cells [dataset 1]

乳腺癌耐药性

冷等离子体

Paclitaxel is a widely used therapeutic chemical for breast cancer treatment, however, cancer recurrence remains obstacle for improved prognosis of cancer patients. In the current study, cold atmosphe

NIAID Data Ecosystem10

Additional file 9: Table S3. of The sialyl-glycolipid stage-specific embryonic antigen 4 marks a subpopulation of chemotherapy-resistant breast cancer cells with mesenchymal features

乳腺癌耐药性

微阵列基因表达

Results of microarray analysis. Results of gene and miRNA expression profiling, Gene Ontology analysis, and miRNA target prediction. (XLSX 118 kb)

NIAID Data Ecosystem20

Enhancer reprogramming driven by high-order assemblies of transcription factors promotes phenotypic plasticity and breast cancer endocrine resistance [ChIP-Seq]. Enhancer reprogramming driven by high-order assemblies of transcription factors promotes phenotypic plasticity and breast cancer endocrine resistance [ChIP-Seq]

乳腺癌耐药性

增强子重编程

Cellular plasticity has emerged as an important mechanism of therapy resistance in cancers, yet the underlying molecular mechanisms remain unclear. Using an established breast cancer cellular model fo

NIAID Data Ecosystem20

Suppression of poised oncogenes by ZMYND8 promotes chemo-sensitization. Suppression of poised oncogenes by ZMYND8 promotes chemo-sensitization

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