Drug-tolerant “persister” cells underlie the emergence of drug-resistant clones and allow residual tumors to survive therapy; thus, represent an attractive therapeutic target to mitigate relapse. Wit

NIAID Data Ecosystem30

Additional file 8 of CDK9 inhibition induces epigenetic reprogramming revealing strategies to circumvent resistance in lymphoma

B细胞淋巴瘤耐药性

表观遗传重编程

Additional file 8.

Figshare2023-03-30 更新20

Chromosomal deletion and chromatin remodeling Drive ABT-199 Resistance in B-cell Lymphomas [ChIP-seq]

B细胞淋巴瘤耐药性

染色质重塑

Drug-tolerant “persister” cells underlie the emergence of drug-resistant clones and allow residual tumors to survive therapy; thus, represent an attractive therapeutic target to mitigate relapse. Wit

NIAID Data Ecosystem10

Additional file 7 of CDK9 inhibition induces epigenetic reprogramming revealing strategies to circumvent resistance in lymphoma

资源简介：

Additional file 7 of CDK9 inhibition induces epigenetic reprogramming revealing strategies to circumvent resistance in lymphoma

资源简介：

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