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Differential Gene Expression and Mitochondrial Dysfunction in Imprinting center deletion (PWS- IC del) Mouse model of Prader-Willi Syndrome

ChEBI2013-04-19 更新2026-07-21 收录

遗传病动物模型

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数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-41759 数据链接链接失效反馈

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Differential Gene Expression and Mitochondrial Dysfunction in Imprinting center deletion (PWS- IC del) Mouse model of Prader-Willi Syndrome

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创建时间：

2013-04-19

自闭症免疫亚型

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Autism spectrum disorder (ASD)7 is associated with multiple physiological abnormalities, including immune dysregulation, and mitochondrial dysfunction. However, an association between these two common

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Alteration of in vivo gene expression in Drosophila intestinal stem cells after OXPHOS disruption and the role of FOXO in it.

干细胞基因表达

线粒体功能障碍

To investigate the in vivo gene expression changes in Drosophila intestinal stem cells (ISCs) when OXPHOS is disrupted and the role of FOXO in regulating this process, we dissected midguts from 4 gr

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Trichostatin A Targets the Mitochondrial Respiratory Chain, Increasing Mitochondrial Reactive Oxygen Species Production to Trigger Apoptosis in Human Breast Cancer Cells

癌症药物机制

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AimHistone deacetylase inhibitors (HDACIs)-based therapies have stimulated interest via their anti-tumor activities, including apoptosis induction, cell cycle arrest, cell differentiation, and autopha

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Caenorhabditis elegans Raw sequence reads. Caenorhabditis elegans

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药物基因表达

This RNAseq data is from wild-type and mitochondrial complex I mutant worms for a nuclear gene-encoded subunit (NDUFS2 homologue), in buffer controls and in mutant young adult worms treated for 24 hou

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Additional file 1 of A bovine model of rhizomelic chondrodysplasia punctata caused by a deep intronic splicing variant in the GNPAT gene

遗传病动物模型

基因组测序

Additional file 1: Table S1. Details of the whole genome sequences used as controls in this study. See the following URLs for more information on the biosample and bioproject IDs: https://www.ncbi.nlm

Figshare2025-05-21 更新10

Differential Gene Expression and Mitochondrial Dysfunction in Imprinting center deletion (PWS- IC del) Mouse model of Prader-Willi Syndrome

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