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Chromatin IP of POF, HP1 and H3K9ac and transcript profiling in S2 cells and salivary glands

ChEBI2007-10-02 更新2026-07-21 收录

染色质免疫沉淀测序

果蝇表观遗传调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-8301 数据链接链接失效反馈

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Chromatin IP of POF, HP1 and H3K9ac and transcript profiling in S2 cells and salivary glands

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创建时间：

2007-10-02

表观遗传检测

染色质免疫沉淀测序

Application of Histone Modification Interacting Domains (HMIDs) as an Alternative to Antibodies. HMIDs and antibodies (taken from ENCODE) were used for precipitation of native chromatin (mononucleosom

NIAID Data Ecosystem40

ChIP-Seq for nuclear GAPDH in ex-vivo cortical microglia from LPS treated mice

染色质免疫沉淀测序

免疫细胞转录组调控

We report the genome wide map for the peaks of occupancy of nuclear GAPDH in cortical microglia from mice treated with LPS. Overall design: Examination of the transcriptional targets that GAPDH can ha

NIAID Data Ecosystem30

Single step cross-linking LCL ChIP-seq. Single step cross-linking LCL ChIP-seq

染色质免疫沉淀测序

ATR-X综合征分子机制

The chromatin remodeller ATRX interacts with the histone chaperone DAXX, to deposit the histone variant H3.3 at sites of nucleosome turnover. ATRX is known to bind repetitive, heterochromatic regions

NIAID Data Ecosystem20

Supplementary Data File

染色质免疫沉淀测序

基因转录调控

Supplementary Data File for "DFF-ChIP: a method to detect and quantify complex interactions between RNA polymerase II, transcription factors, and chromatin"

NIAID Data Ecosystem30

Myc activation in breast cancer is due to p53-loss and sustains self-renewal of cancer stem cells (ChIP-Seq). Mus musculus

乳腺癌分子机制

染色质免疫沉淀测序

Purpose: discover the downstream pathways and the mechanism of target activation by the p53:Myc axis in normal mammary and cancer stem cells Methods: we performed ChIPseq experiments from NMuMG cells

NIAID Data Ecosystem30

Chromatin IP of POF, HP1 and H3K9ac and transcript profiling in S2 cells and salivary glands

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