官方服务:
资源简介:
To explore the level of CCL2/8 levels in clinical samples
应用场景:
创建时间:
2025-12-07
相关数据集
Excessive collagen type VII mediates pleural fibrosis via increasing extracellular matrix stiffness
Cell extracellular matrix interactions are critical in fibrosis. We found that collagen type VII, an ECM component essential for cell anchoring, was upregulated in pleural tissue of patients with tube
NIAID Data Ecosystem80
CXCL8/CXCR2 signaling is Enriched in Fibrotic Myeloproliferative Neoplasms and is Vulnerable to Therapeutic Inhibition [ATAC-Seq]. CXCL8/CXCR2 signaling is Enriched in Fibrotic Myeloproliferative Neoplasms and is Vulnerable to Therapeutic Inhibition [ATAC-Seq]
Pro-inflammatory signaling is a hallmark feature of human cancer, including in myeloproliferative neoplasms (MPNs), most notably myelofibrosis (MF). Dysregulated inflammatory signaling contributes to
NIAID Data Ecosystem30
Table6_Network Pharmacological Analysis and Experimental Validation of the Mechanisms of Action of Si-Ni-San Against Liver Fibrosis.XLSX
Background: Si-Ni-San (SNS), a commonly used traditional Chinese medicine (TCM) formula, has potency against liver diseases, such as hepatitis and non-alcoholic fatty liver disease (NAFLD). However, t
NIAID Data Ecosystem30
Transthyretin alters cardiac fibroblast structure, function and inflammatory gene expression.
Age-related wild type transthyretin amyloidosis (wtATTR) is characterized by systemic deposition of amyloidogenic fibrils of misfolded transthyretin (TTR) in the connective tissue of many organs. In t
Alliance of Genome Resources40
DataSheet_2_Huangkui Capsule Ameliorates Renal Fibrosis in a Unilateral Ureteral Obstruction Mouse Model Through TRPC6 Dependent Signaling Pathways.docx
Renal fibrosis is the final common pathological manifestation of almost all progressive chronic kidney diseases (CKD). Transient receptor potential canonical (TRPC) channels, especially TRPC3/6, were
NIAID Data Ecosystem30



